Patho-Histological Findings of Annular Rupture Related to Left Ventricular Outflow Tract (LVOT) Calcification Following Transcatheter Aortic Valve Replacement (TAVR)
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Ankur Kalra1, Wesley R. Pedersen1, Shannon M. Mackey-Bojack2, Emily R. Duncanson2, Kevin M. Harris1,3 1Minneapolis Heart Institute at Abbott Northwestern Hospital, Minneapolis, MN, USA2Jesse E. Edwards Registry of Cardiovascular Disease, United Hospital, St. Paul, MN, USA 3Electronic correspondence: kharris@mplsheart.com |
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Peri-aortic hematoma has been recently described as a potentially life-threatening complication following transcatheter aortic valve replacement (TAVR). Patient- and procedure-related factors exist that predispose to peri-aortic hematoma formation, which can progress to myocardial rupture at the aortic root-myocardial junction. While conservative therapy with blood pressure control is the expectant management following peri-aortic hematoma |
formation, myocardial rupture can occur at the site of the aortic annulus. Hence, interventionists and echocardiologists must be prepared for emergent intervention to salvage the patient once the complication is recognized. The present report highlights the patho-histological findings related to left ventricular outflow tract calcification following TAVR. The Journal of Heart Valve Disease 2016;25:296-300 |
| Patho-Histological Findings of Annular Rupture Related to Left Ventricular Outflow Tract (LVOT) Calcification Following Transcatheter Aortic Valve Replacement (TAVR) | |
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